Submission Details

Submitter:

Classification:
Moderate
GENCC:100003
Gene:
Disease:
IL10-related early-onset inflammatory bowel disease
Mode Of Inheritance:
Autosomal recessive
Evaluated Date:
02/18/2025
Evidence/Notes:

IL10, also known as CSIF, was first reported in relation to autosomal recessive early-onset inflammatory bowel disease (IBD) in 2010 (Glocker EO, et al., PMID: 20934598). Individuals reported to date with IL10-associated early-onset IBD have presented with IBD with an onset between 1 month and 5 years of age without additional characteristic features.

Three variants (missense) that have been reported in four unrelated probands in three publications (PMIDs: 20934598, 22549091, 29248579) are included in this curation. The mechanism of pathogenicity appears to be loss of function. This gene-disease relationship is also supported by animal models, expression studies, and in vitro functional assays (PMIDs: 8770874, 9312047, 15534372, 1578140, 17137799, 1827484, 22549091, 23158016). IL10 is a well-established anti-inflammatory cytokine (PMID: 1827484, 1578140, 33691712). In vitro experiments have demonstrated that mutant IL10 carrying patient-associated variants displayed impaired suppression of TNFa production by stimulated PBMCs (PMID: 22549091, 23158016). Multiple mouse models of IL10 deficiency, including IL10-knockout and CD4-specific IL10 knockout mice, recapitulate the spontaneous intestinal inflammation in human disease (PMID: 8770874, 15534372). Notably, both IL10Ra and IL10RB, the subunits of the receptor with which IL10 directly interacts (PMID: 9312047), are also associated with early-onset IBD (PMID: 39795980).

In summary, there is moderate evidence to support this gene-disease relationship. While more evidence is needed to establish this relationship definitively, no convincing contradictory evidence has emerged.

PubMed IDs:
1578140 1827484 8770874 9312047 15534372 17137799 20934598 22549091 29248579
Public Report:
Assertion Criteria:
Submitter Submitted Date:
12/05/2025

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